Abstract
Halogenated diarylacetylenes that possess fluorine or chlorine substituents in one aryl ring and N-methylamino or N,N-dimethylamino in the other aryl ring inhibit the proliferation of LS174T colon cancer cells through the repression of c-myc expression and induction of the cyclin-dependent kinase inhibitor-1 (i.e., p21(Wif1/Cip1)) and represent potentially useful antineoplastic agents.
Keywords:
Antineoplastic agents; Colon cancer; Diarylacetylenes; c-myc.
Copyright © 2014 Elsevier Ltd. All rights reserved.
Publication types
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Research Support, N.I.H., Extramural
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Research Support, Non-U.S. Gov't
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Research Support, U.S. Gov't, Non-P.H.S.
MeSH terms
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Alkynes / chemistry*
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Antineoplastic Agents / chemical synthesis
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Antineoplastic Agents / chemistry
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Antineoplastic Agents / pharmacology*
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Cell Line, Tumor
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Cell Proliferation / drug effects
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Dose-Response Relationship, Drug
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Drug Screening Assays, Antitumor
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Halogenation
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Humans
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Hydrocarbons, Chlorinated / chemical synthesis
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Hydrocarbons, Chlorinated / chemistry
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Hydrocarbons, Chlorinated / pharmacology*
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Hydrocarbons, Fluorinated / chemical synthesis
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Hydrocarbons, Fluorinated / chemistry
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Hydrocarbons, Fluorinated / pharmacology*
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Models, Molecular
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Molecular Structure
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Proto-Oncogene Proteins c-myc / antagonists & inhibitors*
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Proto-Oncogene Proteins c-myc / biosynthesis
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Structure-Activity Relationship
Substances
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Alkynes
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Antineoplastic Agents
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Hydrocarbons, Chlorinated
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Hydrocarbons, Fluorinated
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Proto-Oncogene Proteins c-myc