Evaluation of anti-PLA2R1 as measured by a novel ELISA in patients with idiopathic membranous nephropathy: a cohort study

Am J Clin Pathol. 2014 Jul;142(1):29-34. doi: 10.1309/AJCP8QMOY5GLRSFP.

Abstract

Objectives: Autoantibodies against the M-type phospholipase A2 receptor 1 (anti-PLA2R1) have been demonstrated to be very specific for idiopathic membranous nephropathy (MN). We studied a novel enzyme-linked immunosorbent assay (ELISA) and compared results with results obtained using an indirect immunofluorescence (IIF) and a Western blotting test (WB).

Methods: One-hundred nine patients with idiopathic MN were recruited between November 1979 and March 2011. The control cohort comprised serum samples from patients with secondary MN (n = 16) and nephrotic controls (n = 17). The presence of anti-PLA2R1 in serum samples obtained at the time of renal biopsy was determined using ELISA, IIF, and WB.

Results: With similar specificity (≥ 97%), sensitivity varied from 68% (IIF) to 72% (ELISA, WB). Remarkably, patients who were seronegative for anti-PLA2R1 more often entered spontaneous remission (P = .038), whereas seropositive patients were more frequently treated with immunosuppressive agents (P < .001).

Conclusions: ELISA performs excellently in differentiating idiopathic from secondary MN. Furthermore, ELISA shared high agreement with WB and IIF.

Keywords: ELISA; Membranous nephropathy; Nephrotic syndrome; Phospholipase A2 receptor 1; Sensitivity and specificity.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Autoantibodies / blood*
  • Cohort Studies
  • Enzyme-Linked Immunosorbent Assay / methods*
  • Female
  • Glomerulonephritis, Membranous / blood
  • Glomerulonephritis, Membranous / diagnosis*
  • Glomerulonephritis, Membranous / immunology
  • Humans
  • Male
  • Middle Aged
  • Receptors, Phospholipase A2 / immunology*
  • Sensitivity and Specificity

Substances

  • Autoantibodies
  • PLA2R1 protein, human
  • Receptors, Phospholipase A2