Effect of interleukin 1 beta on transducing mechanisms in 235-1 clonal pituitary cells. Part II: Modulation of calcium fluxes

Biochem Biophys Res Commun. 1988 Sep 30;155(3):1097-104. doi: 10.1016/s0006-291x(88)81253-1.

Abstract

In the present study we investigated the effect of the interleukin 1 beta on intracellular free calcium concentrations in 235-1 cell line both in basal conditions and after stimulation by the calcium channel activator maitotoxin. Interleukin 1 beta (from 0.01 pM to 10 nM) was unable to significantly affect basal cytosolic free calcium levels in acute conditions. The preincubation of these cells with interleukin 1 beta for 48h modulates maitotoxin stimulation of calcium fluxes without modifying basal intracellular free calcium levels. Low concentrations of interleukin 1 beta (0.01 pM, 1 pM) caused a marked reduction of intracellular free calcium concentrations increase induced by maitotoxin while higher doses of the monokine potentiated maitotoxin stimulation of calcium fluxes. The specificity of interleukin 1 beta effect was tested by means of polyclonal anti-interleukin 1 beta antibody (titer 1:100) which significantly abolished the inhibitory effect of interleukin 1 beta on free cytosolic calcium levels. These results show that a long lasting interaction of interleukin 1 beta with its receptor is able to influence voltage-sensitive calcium channels activation induced by maitotoxin in 235-1 cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Calcium / metabolism*
  • Clone Cells
  • Drug Synergism
  • Interleukin-1 / pharmacology*
  • Ion Channels / drug effects*
  • Ion Channels / metabolism
  • Marine Toxins / pharmacology
  • Oxocins*
  • Pituitary Gland / drug effects*
  • Pituitary Gland / metabolism
  • Rats

Substances

  • Interleukin-1
  • Ion Channels
  • Marine Toxins
  • Oxocins
  • maitotoxin
  • Calcium