Lack of hepatitis B virus reactivation after anti-tumour necrosis factor treatment in potential occult carriers with chronic inflammatory arthropathies

Eur J Intern Med. 2014 Jun;25(5):482-4. doi: 10.1016/j.ejim.2013.11.014. Epub 2014 Feb 2.

Abstract

Background: Hepatitis B virus (HBV) reactivation in patients positive for antibody to HB core antigen (anti-HBc), negative for HB surface antigen (HBsAg) and HBV-DNA (potential occult HBV carriers), treated with anti-tumor necrosis factor (TNF)α, is a debated question. The aim of the study was to evaluate the safety of anti-TNFα therapy in anti-HBc positive/HBsAg negative subjects with rheumatoid arthritis (RA) and spondyloarthropathy (SpA).

Methods: All consecutive HBsAg negative RA and SpA outpatients referring to the Immuno-Rheumatology Institute at the S. Andrea hospital, Sapienza, University of Rome who had to undergo anti-TNFα therapy.

Results: Among the 169 enrolled subjects, 20 (12%) were potential occult HBV carriers (anti-HBc positive, HBsAg and HBV-DNA negative patients with or without anti-HBs). During the follow-up (mean ± SD 45 ± 22 months), aminotransferases and HBV-DNA, tested every two and six months respectively, did not change.

Conclusion: This study confirms the substantial safety of anti-TNFα therapy in potential occult HBV carriers RA and SpA patients.

Keywords: Anti-TNFα; HBV reactivation; HBcAb-positive; Rheumatic diseases.

MeSH terms

  • Aged
  • Antirheumatic Agents / immunology
  • Antirheumatic Agents / pharmacology*
  • Antirheumatic Agents / therapeutic use
  • Arthritis, Psoriatic / drug therapy
  • Arthritis, Psoriatic / immunology
  • Arthritis, Rheumatoid / drug therapy
  • Arthritis, Rheumatoid / immunology
  • Carrier State / blood
  • Chronic Disease
  • Female
  • Hepatitis B Antibodies / blood
  • Hepatitis B Surface Antigens / blood
  • Hepatitis B virus / physiology*
  • Humans
  • Immunosuppressive Agents / pharmacology*
  • Immunosuppressive Agents / therapeutic use
  • Male
  • Middle Aged
  • Spondylarthropathies / drug therapy*
  • Spondylarthropathies / immunology
  • Spondylitis, Ankylosing / drug therapy
  • Tumor Necrosis Factor-alpha / antagonists & inhibitors*
  • Virus Activation / drug effects*

Substances

  • Antirheumatic Agents
  • Hepatitis B Antibodies
  • Hepatitis B Surface Antigens
  • Immunosuppressive Agents
  • Tumor Necrosis Factor-alpha