Effects of phenobarbital and 1,4-bis[2-(3,5-dichloropyridyloxy)]benzene on differentiated functions in mouse liver

Chem Biol Interact. 1987;64(1-2):83-92. doi: 10.1016/0009-2797(87)90062-7.

Abstract

The promoters of murine hepatocarcinogenesis phenobarbital (PB) and 1,4-bis[2-(3,5-dichloropyridyloxy)]benzene (TCPOBOP) given to adult C3Hf female mice increased the content of total liver DNA by 1.6-1.8-fold each week after the beginning of treatment. Both compounds increased the aminopyrine-N-demethylase activity, decreased the glucose 6-phosphatase (G6Pase), alkaline phosphodiesterase I and alkaline phosphatase specific activities, but did not modify the gamma-glutamyltransferase levels. Both compounds decreased the abundance of tyrosine aminotransferase- and metallothionein I-related RNA transcripts. These findings confirmed the PB-like activity of TCPOBOP and showed that both chemicals had a pleiotropic effect on mouse liver, that was not limited to stimulation of drug metabolism, but also affected other hepatocyte functions.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Differentiation / drug effects
  • Female
  • Glucocorticoids / pharmacology
  • Glucose-6-Phosphatase / analysis
  • Liver / drug effects*
  • Liver / pathology
  • Metallothionein / genetics
  • Mice
  • Mice, Inbred C3H
  • Phenobarbital / toxicity*
  • Pyridines / toxicity*
  • RNA / analysis
  • Transcription, Genetic / drug effects
  • Tyrosine Transaminase / genetics
  • gamma-Glutamyltransferase / analysis

Substances

  • Glucocorticoids
  • Pyridines
  • RNA
  • 1,4-bis(2-(3,5-dichloropyridyloxy))benzene
  • Metallothionein
  • gamma-Glutamyltransferase
  • Tyrosine Transaminase
  • Glucose-6-Phosphatase
  • Phenobarbital