TNFα-induced miR-130 resulted in adipocyte dysfunction during obesity-related inflammation

FEBS Lett. 2013 Nov 29;587(23):3853-8.

Abstract

Adipocytes are continuously stimulated by proinflammatory cytokines such as TNFα, which cause adipocyte dysfunction by facilitating the inflammatory response. Although miR-130 was reported to be an important regulator of adipogenesis by targeting PPARγ mRNA, little is known about the mechanisms regulating miR-130 expression during the proinflammatory response. Here, we examined miR-130 levels in white adipose tissue (WAT) from high-fat diet (HFD) mice and TNFα-stimulated adipocytes. Primary transcripts of miR-130 were increased after TNFα stimulation, indicating that induction of miR-130 during the pro-inflammatory response is regulated by a transcriptional event. A chromatin immunoprecipitation assay showed that p65 binding to the promoter regions of miR-130 was enhanced after TNFα treatment. Taken together, our findings suggest that induction of miR-130 by TNFα is responsible for adipocyte dysfunction.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adipocytes / drug effects
  • Adipocytes / metabolism*
  • Animals
  • Diet, High-Fat / adverse effects
  • Inflammation / etiology
  • Inflammation / metabolism*
  • Mice
  • Mice, Inbred C57BL
  • MicroRNAs / genetics
  • MicroRNAs / metabolism*
  • NIH 3T3 Cells
  • Obesity / complications
  • Obesity / etiology
  • Obesity / metabolism*
  • PPAR gamma / metabolism
  • Transcription, Genetic
  • Tumor Necrosis Factor-alpha / pharmacology*
  • Up-Regulation

Substances

  • MIRN130 microRNA, mouse
  • MicroRNAs
  • PPAR gamma
  • Tumor Necrosis Factor-alpha