Recovery of a chemically synthesized Japanese encephalitis virus reveals two critical adaptive mutations in NS2B and NS4A

J Gen Virol. 2014 Apr;95(Pt 4):806-815. doi: 10.1099/vir.0.061838-0. Epub 2013 Dec 20.

Abstract

A full-length genome infectious clone is a powerful tool for functional assays in virology. In this study, using a chemical synthesized complete genome of Japanese encephalitis virus (JEV) strain SA14 (GenBank accession no. U14163), we constructed a full-length genomic cDNA clone of JEV. The recovered virus from the cDNA clone replicated poorly in baby hamster kidney (BHK-21) cells and in suckling mice brain. Following serial passage in BHK-21 cells, adaptive mutations within the NS2B and NS4A proteins were recovered in the passaged viruses leading to viruses with a large-plaque phenotype. Mutagenesis analysis, using a genome-length RNA and a replicon of JEV, demonstrated that the adaptive mutations restored replication to different degrees, and the restoration efficiencies were in the order: NS2B-T102M<NS4A-R79K<NS2B-T102M+NS4A-R79K. An in vivo virulence assay in mice showed that the recombinant virus containing double mutations showed similar virulence to the WT SA14 (GenBank accession no. M55506). This study reports the first chemically synthesized JEV. A reverse genetics assay demonstrated that substitutions of NS2B-T102M and NS4A-R79K altered JEV replication.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adaptation, Biological*
  • Animals
  • Brain / virology
  • Cell Line
  • Cricetinae
  • DNA Mutational Analysis
  • Disease Models, Animal
  • Encephalitis Virus, Japanese / genetics
  • Encephalitis Virus, Japanese / physiology*
  • Encephalitis, Japanese / pathology
  • Encephalitis, Japanese / virology
  • Female
  • Mice
  • Mice, Inbred BALB C
  • Mutant Proteins / genetics
  • Mutant Proteins / metabolism
  • Mutation, Missense*
  • Reverse Genetics
  • Serial Passage*
  • Viral Nonstructural Proteins / genetics*
  • Viral Nonstructural Proteins / metabolism
  • Virulence
  • Virus Replication*

Substances

  • Mutant Proteins
  • NS2B protein, flavivirus
  • NS4A protein, flavivirus
  • Viral Nonstructural Proteins