Genomic editing opens new avenues for zebrafish as a model for neurodegeneration

J Neurochem. 2013 Nov;127(4):461-70. doi: 10.1111/jnc.12460. Epub 2013 Oct 21.

Abstract

Zebrafish has become a popular model organism to study human diseases. We will highlight the advantages and limitations of zebrafish as a model organism to study neurodegenerative diseases and introduce zinc finger nucleases, transcription activator-like effector nucleases, and the recently established clustered regularly interspaced short palindromic repeats/clustered regularly interspaced short palindromic repeat-associated system for genome editing. The efficiency of the novel genome editing tools now greatly facilitates knock-out and, importantly, also makes knock-in approaches feasible in zebrafish. Genome editing in zebrafish avoids unspecific phenotypes caused by off-target effects and toxicity as frequently seen in conventional knock-down approaches.

Keywords: CRISPR/Cas (clustered regularly interspaced short palindromic repeats); TALEN; genomics; neurodegeneration; zebrafish.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Animals, Genetically Modified
  • Clustered Regularly Interspaced Short Palindromic Repeats
  • Deoxyribonucleases / genetics
  • Disease Models, Animal*
  • Gene Knock-In Techniques
  • Gene Knockout Techniques
  • Genetic Engineering / methods*
  • Genome*
  • Humans
  • Neurodegenerative Diseases / genetics*
  • Zebrafish / genetics*
  • Zinc Fingers

Substances

  • Deoxyribonucleases