Triphenyltin chloride induces spindle microtubule depolymerisation and inhibits meiotic maturation in mouse oocytes

Reprod Fertil Dev. 2014 Oct;26(8):1084-93. doi: 10.1071/RD12332.

Abstract

Meiosis produces haploid gametes for sexual reproduction. Triphenyltin chloride (TPTCL) is a highly bioaccumulated and toxic environmental oestrogen; however, its effect on oocyte meiosis remains unknown. We examined the effect of TPTCL on mouse oocyte meiotic maturation in vitro and in vivo. In vitro, TPTCL inhibited germinal vesicle breakdown (GVBD) and first polar body extrusion (PBE) in a dose-dependent manner. The spindle microtubules completely disassembled and the chromosomes condensed after oocytes were exposed to 5 or 10μgmL(-1) TPTCL. γ-Tubulin protein was abnormally localised near chromosomes rather than on the spindle poles. In vivo, mice received TPTCL by oral gavage for 10 days. The general condition of the mice deteriorated and the ovary coefficient was reduced (P<0.05). The number of secondary and mature ovarian follicles was significantly reduced by 10mgkg(-1) TPTCL (P<0.05). GVBD decreased in a non-significant, dose-dependent manner (P>0.05). PBE was inhibited with 10mgkg(-1) TPTCL (P<0.05). The spindles of in vitro and in vivo metaphase II oocytes were disassembled with 10mgkg(-1) TPTCL. These results suggest that TPTCL seriously affects meiotic maturation by disturbing cell-cycle progression, disturbing the microtubule cytoskeleton and inhibiting follicle development in mouse oocytes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actins / metabolism
  • Animals
  • Cell Cycle Checkpoints / drug effects
  • Cells, Cultured
  • Chromosome Segregation / drug effects
  • Dose-Response Relationship, Drug
  • Female
  • Meiosis / drug effects*
  • Metaphase / drug effects
  • Mice, Inbred ICR
  • Microtubules / drug effects*
  • Microtubules / metabolism
  • Microtubules / pathology
  • Oocytes / drug effects*
  • Oocytes / metabolism
  • Oocytes / pathology
  • Organotin Compounds / toxicity*
  • Polar Bodies / drug effects
  • Polar Bodies / metabolism
  • Polar Bodies / pathology
  • Spindle Apparatus / drug effects*
  • Spindle Apparatus / metabolism
  • Spindle Apparatus / pathology
  • Time Factors
  • Tubulin / metabolism

Substances

  • Actins
  • Organotin Compounds
  • Tubulin
  • triphenyltin chloride