Abstract
A new mutation in mabA, Thr4Ile, was identified in a Mycobacterium tuberculosis isolate from a patient whose culture remained positive after treatment. The same mutation was found in another 5 patients infected by different strains. A putative role for this mutation in the process of diminishing susceptibility to isoniazid is evaluated.
Keywords:
Isoniazid; Mutation; Resistance; mabA.
Copyright © 2013 Elsevier Ltd. All rights reserved.
Publication types
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Case Reports
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Research Support, Non-U.S. Gov't
MeSH terms
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3-Oxoacyl-(Acyl-Carrier-Protein) Reductase / genetics*
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3-Oxoacyl-(Acyl-Carrier-Protein) Reductase / physiology
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Antitubercular Agents / pharmacology*
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Antitubercular Agents / therapeutic use
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Base Sequence
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Drug Resistance, Bacterial / genetics
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Humans
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Isoniazid / pharmacology*
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Isoniazid / therapeutic use
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Male
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Microbial Sensitivity Tests / methods
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Middle Aged
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Molecular Sequence Data
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Mutation*
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Mycobacterium tuberculosis / drug effects
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Mycobacterium tuberculosis / enzymology
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Mycobacterium tuberculosis / genetics*
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Sequence Alignment
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Tuberculosis, Pulmonary / drug therapy
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Tuberculosis, Pulmonary / microbiology
Substances
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Antitubercular Agents
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3-Oxoacyl-(Acyl-Carrier-Protein) Reductase
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Isoniazid