Abstract
Exercise promotes cardioprotection in both humans and animals not only by reducing risk factors associated with cardiovascular disease but by reducing myocardial infarction and improving survival following ischemia. This article will define the role that nitric oxide and β-adrenergic receptors play in mediating the cardioprotective effects of exercise in the setting of ischemia-reperfusion injury.
Publication types
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Research Support, N.I.H., Extramural
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Research Support, Non-U.S. Gov't
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Review
MeSH terms
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Animals
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Cardiovascular Diseases / physiopathology
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Cardiovascular Diseases / prevention & control*
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Disease Models, Animal
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Dogs
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Exercise / physiology*
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Humans
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Mice
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Myocardial Infarction / physiopathology
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Myocardial Infarction / prevention & control
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Nitric Oxide / physiology*
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Physical Conditioning, Animal / physiology
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Rats
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Receptors, Adrenergic, beta / physiology*
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Reperfusion Injury / physiopathology
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Reperfusion Injury / prevention & control
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Signal Transduction / physiology*
Substances
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Receptors, Adrenergic, beta
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Nitric Oxide