Anti-tumor effects of the oral administration of the streptococcal preparation OK-432 (PICIBANIL)--the inhibition of carcinogenesis and growth in rats with ENNG-induced gastrointestinal tumors

Jpn J Surg. 1990 May;20(3):316-26. doi: 10.1007/BF02470667.

Abstract

We examined whether the Streptococcal preparation OK-432, an immunopotentiating agent, increases immunocompetence of the gut-associated lymphoid system (GALS), inhibits gastrointestinal carcinogenesis, and has an anti-tumor effect. 14C-labelled OK-432 was orally and intraperitoneally administered to rats, and the distribution of the agent in various organs then serially evaluated. The concentration of OK-432 in Peyer's patches and mesenteric lymph nodes was higher after oral administration than after intraperitoneal administration, and showed a biphasic pattern peaking at 30 minutes and 5 hours following administration, in the Peyer's patches. With regard to immunocompetence, PHA- and PWM-stimulated blastogenesis of lymphocytes derived from the mesenteric lymph nodes and peripheral blood enhanced, and the helper/suppressor T-cell ratio was elevated after the oral administration of OK-432. Moreover, chemotactic activity of peritoneal macrophages was also increased. ENNG-induced gastrointestinal carcinogenesis was observed in 60 per cent of the rats orally administered OK-432 as compared with 88 per cent of the controls. The 13-month survival rate of the rats with gastrointestinal cancer was 50 per cent in those administered OK-432 as compared with 25 per cent in those administered OK-432 as compared with 25 per cent in the controls. When administered orally, the agent prevented reduction in immuno-competence in the course of carcinogenesis, suppressed carcinogenesis, and prolonged the survival of animals with cancer without any of the side effects associated with injection. The oral administration of OK-432 is thus considered to be an effective non-specific immunotherapy against gastro-intestinal malignancies.

MeSH terms

  • Administration, Oral
  • Animals
  • Biological Products / therapeutic use*
  • Carcinogens / toxicity
  • Digestive System / immunology
  • Gastrointestinal Neoplasms / chemically induced
  • Gastrointestinal Neoplasms / therapy*
  • Immunocompetence
  • Lymphoid Tissue / immunology
  • Macrophages / immunology
  • Male
  • Methylnitronitrosoguanidine / toxicity
  • Picibanil / administration & dosage
  • Picibanil / therapeutic use*
  • Rats
  • Rats, Inbred Strains
  • T-Lymphocytes / immunology

Substances

  • Biological Products
  • Carcinogens
  • Methylnitronitrosoguanidine
  • Picibanil
  • ENNG