Hamster-adapted Sin Nombre virus causes disseminated infection and efficiently replicates in pulmonary endothelial cells without signs of disease

J Virol. 2013 Apr;87(8):4778-82. doi: 10.1128/JVI.03291-12. Epub 2013 Feb 6.

Abstract

To date, a laboratory animal model for the study of Sin Nombre virus (SNV) infection or associated disease has not been described. Unlike infection with Andes virus, which causes lethal hantavirus pulmonary syndrome (HPS)-like disease in hamsters, SNV infection is short-lived, with no viremia and little dissemination. Here we investigated the effect of passaging SNV in hamsters. We found that a host-adapted SNV achieves prolonged and disseminated infection in hamsters, including efficient replication in pulmonary endothelial cells, albeit without signs of disease.

Publication types

  • Research Support, N.I.H., Intramural

MeSH terms

  • Adaptation, Biological
  • Animals
  • Asymptomatic Diseases
  • Cricetinae
  • Disease Models, Animal*
  • Endothelial Cells / virology*
  • Hantavirus Pulmonary Syndrome / veterinary*
  • Hantavirus Pulmonary Syndrome / virology
  • Sin Nombre virus / growth & development
  • Sin Nombre virus / isolation & purification
  • Sin Nombre virus / pathogenicity*
  • Virus Replication*