Radiation-induced bone marrow apoptosis, inflammatory bystander-type signaling and tissue cytotoxicity

Int J Radiat Biol. 2013 Mar;89(3):139-46. doi: 10.3109/09553002.2013.741280. Epub 2012 Nov 6.

Abstract

Purpose: A study of irradiated (0.25-2 Gy) murine bone marrow has investigated the relationships between apoptotic responses of cells exposed in vivo and in vitro and between in vivo apoptosis and tissue cytotoxicity.

Materials and methods: The time course of reduction in bone marrow cellularity in vivo was determined by femoral cell counts and apoptosis measurements obtained using three commonly used assays. Inflammatory pro-apoptotic cytokine production at 24 h post-exposure in vivo was investigated using a bystander protocol.

Results: In vivo, there is a dose- and time-dependent non-linear reduction in bone marrow cellularity up to 24 h post- irradiation not directly represented by apoptosis measurements. The majority of cells are killed within 6 h but there is on-going cell loss in vivo up to 24 h post-irradiation in the absence of elevated levels of apoptosis and associated with the induction of cytokines produced in response to the initial tumor protein 53 (p53)-dependent apoptosis.

Conclusion: The results demonstrate that small increases in measured apoptosis can reflect significant intramedullary cell death and with apoptotic processes being responsible for pro-inflammatory mechanisms that can contribute to additional on-going cell death. The findings demonstrate the importance of studying tissue responses when considering the mechanisms underlying the consequences of radiation exposures.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Apoptosis / radiation effects*
  • Bone Marrow Cells / immunology
  • Bone Marrow Cells / pathology*
  • Bone Marrow Cells / radiation effects*
  • Bystander Effect / radiation effects
  • Cytokines / biosynthesis
  • Cytotoxicity, Immunologic / radiation effects
  • Dose-Response Relationship, Radiation
  • Gamma Rays / adverse effects
  • Genes, p53
  • Inflammation Mediators / metabolism
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Mice, Inbred CBA
  • Mice, Knockout
  • Signal Transduction / immunology
  • Signal Transduction / radiation effects
  • Translational Research, Biomedical

Substances

  • Cytokines
  • Inflammation Mediators