Stimulation of human EBV- and CMV-specific cytolytic effector function using allogeneic HLA molecules

J Immunol. 2012 Nov 15;189(10):4825-31. doi: 10.4049/jimmunol.1201034. Epub 2012 Oct 17.

Abstract

Viral infection is a major cause of morbidity and mortality, and there are few therapeutic options available to augment a virus-specific T cell response. Although allo-HLA cross-reactivity from virus-specific memory T cells is common, it is unclear whether priming with specific allogeneic cells could conversely elicit a viral peptide/self-HLA restricted cytotoxic T cell response in humans. First, we used the previously described allo-HLA-B*44:02 cross-reactivity of EBV peptide/HLA-B8 restricted T cells, to determine whether allogeneic HLA stimulation can elicit a cytolytic immune response against EBV. HLA-B8(+) HLA-B44(-) EBV-seropositive PBMCs were stimulated with either HLA-B*44:02(+) or HLA-B*44:03(+) mismatched irradiated PBMCs in a 7-10 d MLR. The allo-HLA stimulated responder cells were then evaluated for cytotoxicity using EBV peptide loaded autologous target cells and unloaded HLA-B8(+) EBV LCL target cells. PBMCs from EBV-seropositive donors gained EBV-specific cytolytic effector function following specific allo-HLA stimulation. Finally, we also elicited cytolytic CMV-specific responses using specific allogeneic cell stimulation, to confirm that this technique can be used to elicit viral peptide/self-HLA restricted responses even from nonpublic TCR responses. Allogeneic cell stimulation used as a cell therapy may be a potential tool to augment an antiviral T cell response in patients with EBV or CMV infection.

Publication types

  • Clinical Trial

MeSH terms

  • Cells, Cultured
  • Cytomegalovirus / immunology*
  • Cytomegalovirus Infections / immunology*
  • Epstein-Barr Virus Infections / immunology*
  • Female
  • HLA-B Antigens / immunology*
  • Herpesvirus 4, Human / immunology*
  • Humans
  • Immunologic Memory*
  • Male
  • Peptides / immunology
  • T-Lymphocytes, Cytotoxic / immunology*
  • Viral Proteins / immunology

Substances

  • HLA-B Antigens
  • Peptides
  • Viral Proteins