Inhibition of autoimmune disease in a murine model of systemic lupus erythematosus induced by exposure to syngeneic photoinactivated lymphocytes

J Invest Dermatol. 1990 Jan;94(1):52-7. doi: 10.1111/1523-1747.ep12873349.

Abstract

MRL/l mice develop progressive, virulent autoimmune disease that has many of the features of systemic lupus erythematosus. Prophylactic treatment of MRL/l mice with syngeneic photoinactivated autoimmune splenocytes improves survival and inhibits the fulminant hyperproliferation of abnormal T cells and the production of high titer anti-DNA antibody invariably found in untreated mice. The proliferation of Thy 1+ splenic T cells was significantly decreased, and prolonged retention of the response to T-cell mitogen was found in treated mice. Treatment with unmodified cells induced a partial inhibition of disease features which did not prolong survival rates. These results suggest that phototherapy potentiates a normal immunoregulatory process which enables suppression of the development of abnormal cell populations in young MRL/l mice with relatively intact immune systems.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Autoantibodies / analysis
  • Autoimmune Diseases / etiology
  • Autoimmune Diseases / mortality
  • Autoimmune Diseases / prevention & control*
  • Concanavalin A / pharmacology
  • DNA / immunology
  • Female
  • Light*
  • Lipopolysaccharides / pharmacology
  • Lupus Erythematosus, Systemic / complications
  • Lupus Erythematosus, Systemic / pathology
  • Lupus Erythematosus, Systemic / therapy*
  • Lymphocyte Activation / radiation effects*
  • Lymphocyte Transfusion*
  • Lymphocytes / drug effects
  • Lymphocytes / radiation effects
  • Lymphoid Tissue / pathology
  • Mice
  • Mice, Inbred Strains
  • Phenotype
  • Spleen / cytology
  • Survival Analysis
  • Transplantation, Isogeneic

Substances

  • Autoantibodies
  • Lipopolysaccharides
  • Concanavalin A
  • DNA