PBK/TOPK expression during TPA-induced HL-60 leukemic cell differentiation

Asian Pac J Cancer Prev. 2012;13(5):2145-8. doi: 10.7314/apjcp.2012.13.5.2145.

Abstract

Objective: This study concerns expression of PBK/TOPK during differentiation of HL-60 leukemic cells induced by tetradecanoyl phorbol acetate (TPA).

Methods: Wright-Giemsa staining was performed to observe morphological changes in the HL-60 cells, and flow cytometry was used to assess the cell cycle and CD11b, CD14, CD13, and CD33 expression. PBK/TOPK levels were determined by Western blot analysis.

Results: After treating HL60 cells with 5.1×10⁻⁹ mmol/L of TPA for three days, the number of nitroblue-tetrazolium-positive cells and CD11b, CD13, and CD14 expression increased, whereas the PBK/TOPK levels decreased.

Conclusions: TPA can inhibit proliferation and induce differentiation of HL60 cells of the granulocytic or monocytic lineage. PBK/TOPK expression was downregulated during this process, whereas the Pho-PBK/TOPK expression was increased.

MeSH terms

  • Antigens, Surface / metabolism
  • Apoptosis / drug effects
  • Blotting, Western
  • Carcinogens / pharmacology*
  • Cell Cycle / drug effects
  • Cell Differentiation / drug effects*
  • Cell Membrane / metabolism
  • Cell Proliferation / drug effects
  • Flow Cytometry
  • Granulocytes / cytology
  • Granulocytes / drug effects
  • Granulocytes / metabolism
  • Humans
  • Leukemia / metabolism
  • Leukemia / pathology*
  • Mitogen-Activated Protein Kinase Kinases / metabolism*
  • Monocytes / cytology
  • Monocytes / drug effects
  • Monocytes / metabolism
  • Tetradecanoylphorbol Acetate / pharmacology*
  • Tumor Cells, Cultured

Substances

  • Antigens, Surface
  • Carcinogens
  • Mitogen-Activated Protein Kinase Kinases
  • PDZ-binding kinase
  • Tetradecanoylphorbol Acetate