Transient receptor potential vanilloid 4 blockade protects against experimental colitis in mice: a new strategy for inflammatory bowel diseases treatment?

Neurogastroenterol Motil. 2012 Nov;24(11):e557-60. doi: 10.1111/j.1365-2982.2012.01999.x. Epub 2012 Aug 10.

Abstract

Recent reports suggested that the activation of Transient Receptor Potential Vanilloid 4 (TRPV4) receptors in the gastrointestinal tract has pro-inflammatory effects. In this study, we demonstrated for the first time that TRPV4 mRNA expression is up-regulated in patients with inflammatory bowel diseases (IBD). Furthermore, selective blockade of TRPV4 in the 2,4,6-trinitrobenzenesulfonic acid animal model alleviates colitis and pain associated with the intestinal inflammation. Our study indicates that TRPV4 may play a role in mechanisms of defense in intestinal inflammation and that TRPV4 may be an attractive target for future systemic or topic anti-inflammatory treatment in patients with IBD.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Disease Models, Animal
  • Gastrointestinal Agents / pharmacology
  • Humans
  • Inflammatory Bowel Diseases / complications
  • Inflammatory Bowel Diseases / metabolism*
  • Mice
  • Pain / etiology
  • Pain / metabolism*
  • Real-Time Polymerase Chain Reaction
  • Reverse Transcriptase Polymerase Chain Reaction
  • Sulfonamides / pharmacology
  • TRPV Cation Channels / antagonists & inhibitors
  • TRPV Cation Channels / metabolism*

Substances

  • Gastrointestinal Agents
  • RN 1734
  • Sulfonamides
  • TRPV Cation Channels
  • TRPV4 protein, human
  • Trpv4 protein, mouse