Genetic variation in the transforming growth factor-β-signaling pathway, lifestyle factors, and risk of colon or rectal cancer

Dis Colon Rectum. 2012 May;55(5):532-40. doi: 10.1097/DCR.0b013e31824b5feb.

Abstract

Background: The transforming growth factor-β-signaling pathway has been identified as being involved in colorectal cancer.

Objective: The aim of this study was to determine how diet and lifestyle factors in combination with genetic variation in the transforming growth factor-β-signaling pathway alters colorectal cancer risk.

Design: We used data from 2 population-based case-control studies.

Patients: Participants included patients with colon cancer (n = 1574) and controls (n = 1970) and patients with rectal cancer ( n = 791) and controls (n = 999).

Main outcome measures: The primary outcomes measured were newly diagnosed cases of colon or rectal cancer.

Results: Colon and rectal cancer risk increased with the number of at-risk genotypes within the transforming growth factor-β-signaling pathway (OR 3.68, 95% CI 2.74,4.94 for colon cancer; OR 3.89, 95% CI 2.66,5.69 for rectal cancer). A high at-risk lifestyle score also resulted in significant increased risk with number of at-risk lifestyle factors (OR 2.99, 95% CI 2.32,3.85 for colon cancer; OR 3.37, 95% CI 2.24,5.07 for rectal cancer). The combination of high-risk genotype and high-risk lifestyle results in the greatest increase in risk (OR 7.89, 95% CI 4.45,13.96 for colon cancer; OR 8.75, 95% CI 3.66,20.89 for rectal cancer).

Limitations: The study results need validation in other large studies of colon and rectal cancer.

Conclusions: In summary, our data suggest that there is increased colon and rectal cancer risk with increasing number of at-risk genotypes and at-risk lifestyle factors. Although the integrity of the pathway can be diminished by a number of high-risk genotypes, this risk can be offset, in part, by maintaining a healthy lifestyle.

Publication types

  • Comparative Study
  • Multicenter Study
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Colonic Neoplasms / epidemiology
  • Colonic Neoplasms / genetics*
  • Colonic Neoplasms / metabolism
  • DNA, Neoplasm / genetics*
  • Female
  • Genetic Variation*
  • Genotype
  • Humans
  • Incidence
  • Life Style*
  • Male
  • Middle Aged
  • Polymerase Chain Reaction
  • Rectal Neoplasms / epidemiology
  • Rectal Neoplasms / genetics*
  • Rectal Neoplasms / metabolism
  • Retrospective Studies
  • Signal Transduction
  • Transforming Growth Factor alpha / genetics*
  • Transforming Growth Factor alpha / metabolism
  • United States / epidemiology

Substances

  • DNA, Neoplasm
  • Transforming Growth Factor alpha