Pseudo-heterozygous rearrangement mutation of parkin

Mov Disord. 2012 Apr;27(4):552-5. doi: 10.1002/mds.24906. Epub 2012 Feb 5.

Abstract

Background: Mutations in parkin are the most frequent cause of autosomal recessive parkinsonism. Quantitative PCR is used to detect parkin rearrangements. However, the method has an inherent problem-deletion and duplication in the same allelic exon could be determined as normal. To present this misidentification, we report a family with compound heterozygous rearrangements in parkin.

Methods: A patient with early-onset parkinsonism and the parents were investigated by quantitative PCR, haplotype analysis, reverse-transcription PCR, and direct sequencing.

Results: A single heterozygous duplication (duplication of exons 6-7) was identified in the patient by quantitative PCR. Detailed analysis of the family revealed the patient carried compound heterozygous of combined deletion (deletion of exons 3-5) and duplication (duplication of exons 3-7).

Conclusions: For correct determination of rearrangement mutation, mutation analysis of the patient as well as other family members and/or break-point analysis of genomic DNA and at the transcript level should be conducted.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • DNA Mutational Analysis
  • Female
  • Humans
  • Mutation*
  • Parkinsonian Disorders / genetics*
  • Ubiquitin-Protein Ligases / genetics*
  • Young Adult

Substances

  • Ubiquitin-Protein Ligases
  • parkin protein