Enhanced differentiation of three-gene-reprogrammed induced pluripotent stem cells into adipocytes via adenoviral-mediated PGC-1α overexpression

Int J Mol Sci. 2011;12(11):7554-68. doi: 10.3390/ijms12117554. Epub 2011 Nov 7.

Abstract

Induced pluripotent stem cells formed by the introduction of only three factors, Oct4/Sox2/Klf4 (3-gene iPSCs), may provide a safer option for stem cell-based therapy than iPSCs conventionally introduced with four-gene iPSCs. Peroxisome proliferator-activated receptor gamma coactivator-1α (PGC-1α) plays an important role during brown fat development. However, the potential roles of PGC-1α in regulating mitochondrial biogenesis and the differentiation of iPSCs are still unclear. Here, we investigated the effects of adenovirus-mediated PGC-1α overexpression in 3-gene iPSCs. PGC-1α overexpression resulted in increased mitochondrial mass, reactive oxygen species production, and oxygen consumption. Microarray-based bioinformatics showed that the gene expression pattern of PGC-1α-overexpressing 3-gene iPSCs resembled the expression pattern observed in adipocytes. Furthermore, PGC-1α overexpression enhanced adipogenic differentiation and the expression of several brown fat markers, including uncoupling protein-1, cytochrome C, and nuclear respiratory factor-1, whereas it inhibited the expression of the white fat marker uncoupling protein-2. Furthermore, PGC-1α overexpression significantly suppressed osteogenic differentiation. These data demonstrate that PGC-1α directs the differentiation of 3-gene iPSCs into adipocyte-like cells with features of brown fat cells. This may provide a therapeutic strategy for the treatment of mitochondrial disorders and obesity.

Keywords: brown adipocyte; c-Myc; induced pluripotent stem cell; peroxisome proliferator-activated receptor gamma coactivator-1α.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenoviridae / genetics
  • Adipocytes / cytology*
  • Animals
  • Cell Differentiation / genetics*
  • Cytochromes c / genetics
  • Cytochromes c / metabolism
  • Induced Pluripotent Stem Cells / cytology*
  • Ion Channels / genetics
  • Ion Channels / metabolism
  • Kruppel-Like Factor 4
  • Kruppel-Like Transcription Factors / genetics
  • Kruppel-Like Transcription Factors / metabolism
  • Mice
  • Mitochondria / genetics
  • Mitochondria / metabolism
  • Mitochondrial Proteins / genetics
  • Mitochondrial Proteins / metabolism
  • Nuclear Respiratory Factor 1 / genetics
  • Nuclear Respiratory Factor 1 / metabolism
  • Octamer Transcription Factor-3 / genetics
  • Octamer Transcription Factor-3 / metabolism
  • Osteogenesis / genetics
  • Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha
  • Reactive Oxygen Species / metabolism
  • SOXB1 Transcription Factors / genetics
  • SOXB1 Transcription Factors / metabolism
  • Transcription Factors / genetics*
  • Transcription Factors / metabolism
  • Uncoupling Protein 1
  • Uncoupling Protein 2
  • Up-Regulation

Substances

  • Ion Channels
  • Klf4 protein, mouse
  • Kruppel-Like Factor 4
  • Kruppel-Like Transcription Factors
  • Mitochondrial Proteins
  • Nrf1 protein, mouse
  • Nuclear Respiratory Factor 1
  • Octamer Transcription Factor-3
  • Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha
  • Pou5f1 protein, mouse
  • Ppargc1a protein, mouse
  • Reactive Oxygen Species
  • SOXB1 Transcription Factors
  • Sox2 protein, mouse
  • Transcription Factors
  • Ucp1 protein, mouse
  • Ucp2 protein, mouse
  • Uncoupling Protein 1
  • Uncoupling Protein 2
  • Cytochromes c