Abstract
Ethionamide (ETH) is a second-line drug for the treatment of tuberculosis. As a prodrug, ETH has to be activated by EthA. ethA is controlled by its repressor EthR. 2-Phenylethyl-butyrate (2-PEB) inhibits EthR binding, enhances expression of EthA, and thereby enhances the growth-inhibitory effects of ethionamide, isoxyl, and thiacetazone in Mycobacterium tuberculosis strains with resistance to ETH due to inhA promoter mutations but not ethA mutations.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Antitubercular Agents / pharmacology*
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Bacterial Proteins / drug effects
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Bacterial Proteins / genetics
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Bacterial Proteins / metabolism
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Butyrates / chemistry
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Butyrates / pharmacology*
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Drug Resistance, Multiple, Bacterial
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Drug Synergism
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Ethionamide / pharmacology*
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Gene Expression Regulation, Bacterial
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Humans
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Microbial Sensitivity Tests
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Mutation
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Mycobacterium tuberculosis / drug effects*
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Mycobacterium tuberculosis / growth & development
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Mycobacterium tuberculosis / isolation & purification
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Oxidoreductases / genetics
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Oxygenases / drug effects
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Oxygenases / genetics
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Oxygenases / metabolism
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Phenylthiourea / analogs & derivatives*
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Phenylthiourea / pharmacology
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Thioacetazone / pharmacology*
Substances
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Antitubercular Agents
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Bacterial Proteins
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Butyrates
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etaA protein, Mycobacterium tuberculosis
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thiocarlide
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Phenylthiourea
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Oxidoreductases
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Oxygenases
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InhA protein, Mycobacterium
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Thioacetazone
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Ethionamide