Abstract
Next generation NNRTIs are sought which possess both broad spectrum antiviral activity against key mutant strains and a high genetic barrier to the selection of new mutant viral strains. Pyridones were evaluated as an acyclic conformational constraint to replace the aryl ether core of MK-4965 (1) and the more rigid indazole constraint of MK-6186 (2). The resulting pyridone compounds are potent inhibitors of HIV RT and have antiviral activity in cell culture that is superior to other next generation NNRTI's.
Copyright © 2011 Elsevier Ltd. All rights reserved.
MeSH terms
-
Binding Sites
-
Cell Line
-
Computer Simulation
-
Drug Design
-
Enzyme Activation / drug effects
-
HIV / enzymology
-
HIV Reverse Transcriptase / antagonists & inhibitors*
-
HIV Reverse Transcriptase / metabolism
-
Humans
-
Protein Structure, Tertiary
-
Pyrazoles / chemistry
-
Pyridines / chemistry
-
Pyridones / chemical synthesis
-
Pyridones / chemistry*
-
Pyridones / pharmacology
-
Reverse Transcriptase Inhibitors / chemical synthesis*
-
Reverse Transcriptase Inhibitors / chemistry
-
Reverse Transcriptase Inhibitors / pharmacology
Substances
-
3-(5-((6-amino-1H-pyrazolo(3,4-b)pyridine-3-yl)methoxy)-2-chlorophenoxy)-5-chlorobenzonitrile
-
Pyrazoles
-
Pyridines
-
Pyridones
-
Reverse Transcriptase Inhibitors
-
HIV Reverse Transcriptase