regulation of the invasion and metastasis of human glioma cells by polypeptide N-acetylgalactosaminyltransferase 2

Mol Med Rep. 2011 Nov-Dec;4(6):1299-305. doi: 10.3892/mmr.2011.569. Epub 2011 Aug 22.

Abstract

Polypeptide, N-acetylgalactosaminyltransferase 2 (ppGalNAc-T2), is a member of the ppGalNAcT family which is the initial glycosyltransferase in O-glycan synthesis. Our previous studies have demonstrated that ppGalNAc-T2 plays a vital role in the process of tumor emergence and development. In this study, we aimed to determine whether ppGalNAc-T2 is correlated with the invasion and metastasis of human glioma cells. PpGalNAc-T2 sense vectors and interference vectors constructed in our laboratory were successfully transfected into SHG44 and U251 cells, which were observed through fluorescence microscopy, RT-PCR and western blot analysis. The metastasis of U251 cells was determined using a wound healing assay, and ppGalNAc-T2 was found to inhibit cell migration. By RT-PCR, glioma cell invasion assay showed that the expression of MMP-2 was increased in ppGalNAc-T2-knockdown cells, while the expression of MMP-9 and TGF-β1 was decreased in ppGalNAc-T2-overexpressing cells at the mRNA level. Meanwhile, the expression of TIMP-2 at the mRNA level was contrary to MMP-2. In summary, our findings indicate that ppGalNAc-T2 plays a poten-tial role in glioma cell migration and invasion, suggesting a new molecular therapeutic target for human malignant glioma treatment.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Brain Neoplasms / metabolism
  • Brain Neoplasms / pathology*
  • Cell Line, Tumor
  • Cell Movement
  • Gene Knockdown Techniques
  • Glioma / metabolism
  • Glioma / pathology*
  • Humans
  • Matrix Metalloproteinase 2 / genetics
  • Matrix Metalloproteinase 2 / metabolism
  • Matrix Metalloproteinase 9 / genetics
  • Matrix Metalloproteinase 9 / metabolism
  • Microscopy, Fluorescence
  • N-Acetylgalactosaminyltransferases / genetics
  • N-Acetylgalactosaminyltransferases / metabolism*
  • Neoplasm Metastasis
  • Plasmids / chemistry
  • Plasmids / metabolism
  • Polypeptide N-acetylgalactosaminyltransferase
  • RNA, Messenger / metabolism
  • Tissue Inhibitor of Metalloproteinase-2 / genetics
  • Tissue Inhibitor of Metalloproteinase-2 / metabolism
  • Transforming Growth Factor beta1 / genetics
  • Transforming Growth Factor beta1 / metabolism

Substances

  • RNA, Messenger
  • Transforming Growth Factor beta1
  • Tissue Inhibitor of Metalloproteinase-2
  • N-Acetylgalactosaminyltransferases
  • Matrix Metalloproteinase 2
  • Matrix Metalloproteinase 9