Dibutyrylcytidine 3',5'-cyclic monophosphate stimulates neurite outgrowth in rat pheochromocytoma PC12

Neurosci Lett. 1990 Jul 13;114(3):323-8. doi: 10.1016/0304-3940(90)90584-v.

Abstract

Dibutyrylcytidine 3',5'-cyclic monophosphate (Bt2cCMP) stimulated neurite outgrowth in rat pheochromocytoma PC12 cells in a dose-dependent manner at the dose range from 0.5 mM to 6 mM. About 25% of the cells had neurites in response to 6 mM Bt2cCMP. In contrast with the effect of nerve growth factor (NGF) which increased the percentage of cells with neurites gradually during a 6-day culture period, the stimulating effect of Bt2cCMP reached a plateau 2 days after plating. Staurosporine inhibited the neurite outgrowth induced by NGF, but not that by Bt2cCMP. These results suggest that Bt2cCMP stimulates neuronal differentiation by a mechanism different from that by NGF.

MeSH terms

  • Adrenal Gland Neoplasms
  • Animals
  • Axons / drug effects
  • Axons / physiology*
  • Cyclic CMP / analogs & derivatives
  • Cyclic CMP / pharmacology*
  • Cytosine Nucleotides / pharmacology*
  • Nerve Growth Factors / pharmacology
  • Pheochromocytoma
  • Rats

Substances

  • Cytosine Nucleotides
  • Nerve Growth Factors
  • Cyclic CMP
  • dibutyryl cyclic-3',5'-cytidine monophosphate