Premature atherosclerosis in non-transfusion-dependent β-thalassemia intermedia

Cardiology. 2011;118(3):159-63. doi: 10.1159/000327997. Epub 2011 Jun 1.

Abstract

Objectives: β-Thalassemias are inherited hemolytic anemias with a broad phenotype and an increased rate of vascular complications despite a near absence of coronary artery disease.

Methods: We investigated the presence of endothelial dysfunction and subclinical atherosclerosis in non-transfusion-dependent patients with β-thalassemia intermedia (β-TI) by means of flow-mediated (FMD) and flow-independent (FID) brachial artery dilatation and carotid artery intima-media thickness.

Results: In 20 nondiabetic young adults with β-TI, FMD (6.6 ± 3.7 vs. 10.3 ± 3.1%; p = 0.002) and FID (14.0 ± 4.7 vs. 18.0 ± 5.6%; p = 0.02) were both lower relative to the values in 20 matched control subjects, whereas the intima-media thickness was increased (0.51 ± 0.09 vs. 0.46 ± 0.07 mm; p = 0.049). Fibrin generation, soluble endothelial activation markers, and proinflammatory proteins were higher in the patient group, while the plasma cholesterol level was lower.

Conclusions: These findings indicate premature atherosclerosis among patients with β-TI; this is in accord with the high incidence of noncoronary vascular episodes in β-TI.

MeSH terms

  • Adult
  • Atherosclerosis / diagnosis
  • Atherosclerosis / etiology*
  • Blood Transfusion
  • Brachial Artery / pathology
  • Brachial Artery / physiopathology
  • Carotid Arteries / pathology
  • Carotid Arteries / physiopathology
  • Case-Control Studies
  • Female
  • Humans
  • Male
  • Middle Aged
  • Tunica Intima / pathology
  • Tunica Intima / physiopathology
  • Tunica Media / pathology
  • Tunica Media / physiopathology
  • Young Adult
  • beta-Thalassemia / complications*
  • beta-Thalassemia / pathology*
  • beta-Thalassemia / physiopathology