Sleep deprivation differentially affects dopamine receptor subtypes in mouse striatum

Neuroreport. 2011 Jul 13;22(10):489-93. doi: 10.1097/WNR.0b013e32834846a0.

Abstract

The effects of sleep deprivation on dopaminergic systems remain elusive, in part due to the lack of selective ligands for dopamine receptor subtypes. We examined D1, D2, and D3 receptor density in the mouse brain after sleep deprivation by receptor autoradiography using [H]SCH 23390 for D1R, [H]raclopride for D2R, and [H]WC-10 for D3R (a novel D3R-selective compound developed in our laboratory, not previously reported in mouse). Sleep-deprived mice showed a significant decrease in D1R, no change in D2R, and a significant increase in D3R binding in striatum. This pattern of dopamine receptor changes was not seen in mice subjected to restraint stress, suggesting specificity to sleep. These data provide evidence that brain dopaminergic circuits are remodeled after sleep deprivation.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Autoradiography / methods
  • Benzazepines / pharmacokinetics
  • Corpus Striatum / drug effects
  • Corpus Striatum / metabolism*
  • Dopamine Agents / pharmacokinetics
  • Female
  • Hydrocarbons, Iodinated / pharmacokinetics
  • Mice
  • Mice, Inbred C57BL
  • Piperazines / pharmacokinetics
  • Protein Binding / drug effects
  • Protein Subunits / metabolism*
  • Receptors, Dopamine / classification
  • Receptors, Dopamine / metabolism*
  • Restraint, Physical / methods
  • Sleep Deprivation / pathology*
  • Tritium / pharmacokinetics

Substances

  • Benzazepines
  • Dopamine Agents
  • Hydrocarbons, Iodinated
  • Piperazines
  • Protein Subunits
  • Receptors, Dopamine
  • SCH 23390
  • Tritium
  • methyl iodide
  • phenylpiperazine