Tri-iodothyronine enhances liver regeneration after living donor liver transplantation in rats

J Hepatobiliary Pancreat Sci. 2011 Nov;18(6):806-14. doi: 10.1007/s00534-011-0397-2.

Abstract

Background: Tri-iodothyronine (T3) has been shown to be a hepatic mitogen. The aim of this study was to evaluate the effect of T3 on liver regeneration after 50% partial liver transplantation (pLT) in rats.

Methods: Immediately after pLT, a single dose of T3 (4 mg/kg body weight) was administered. Liver/body weight ratio (LBWR), hepatocyte proliferation (Ki-67), biochemical parameters, and changes in cell cycle related proteins were evaluated.

Results: T3 promoted liver regeneration as shown by an increased liver/body weight ratio and Ki-67 proliferation index after pLT. On the transcriptional level, T3-treated rats had an increased expression of cyclin D1 and cyclin A as demonstrated by real time RT-PCR and Western blot.

Conclusions: Exogenous administration of T3 significantly improved liver regeneration after pLT, and therefore it may represent a promising strategy to improve the clinical outcome after living donor liver transplantation in the future.

Keywords: T3; cell cycle; hepatic mitogen; liver regeneration; liver transplantation.

MeSH terms

  • Animals
  • Disease Models, Animal
  • Liver Diseases / surgery*
  • Liver Regeneration / drug effects*
  • Liver Transplantation*
  • Living Donors*
  • Male
  • Organ Size / drug effects
  • Rats
  • Rats, Sprague-Dawley
  • Triiodothyronine / analogs & derivatives*
  • Triiodothyronine / pharmacology

Substances

  • Triiodothyronine
  • 3,3',5-triiodothyronamine