AlgW regulates multiple Pseudomonas syringae virulence strategies

Mol Microbiol. 2011 Apr;80(2):364-77. doi: 10.1111/j.1365-2958.2011.07571.x. Epub 2011 Feb 28.

Abstract

Gram-negative bacterial pathogens have evolved a number of virulence-promoting strategies including the production of extracellular polysaccharides such as alginate and the injection of effector proteins into host cells. The induction of these virulence mechanisms can be associated with concomitant downregulation of the abundance of proteins that trigger the host immune system, such as bacterial flagellin. In Pseudomonas syringae, we observed that bacterial motility and the abundance of flagellin were significantly reduced under conditions that induce the type III secretion system. To identify genes involved in this negative regulation, we conducted a forward genetic screen with P. syringae pv. maculicola ES4326 using motility as a screening phenotype. We identified the periplasmic protease AlgW as a key negative regulator of flagellin abundance that also positively regulates alginate biosynthesis and the type III secretion system. We also demonstrate that AlgW constitutes a major virulence determinant of P. syringae required to dampen plant immune responses. Our findings support the conclusion that P. syringae co-ordinately regulates virulence strategies through AlgW in order to effectively suppress host immunity.

MeSH terms

  • Alginates / metabolism
  • Bacterial Proteins / metabolism*
  • Flagellin / metabolism
  • Gene Expression Regulation, Bacterial*
  • Glucuronic Acid / metabolism
  • Hexuronic Acids / metabolism
  • Immune Evasion
  • Locomotion
  • Plant Diseases / microbiology
  • Plants / immunology
  • Pseudomonas syringae / genetics
  • Pseudomonas syringae / pathogenicity
  • Pseudomonas syringae / physiology*
  • Repressor Proteins / metabolism*
  • Virulence
  • Virulence Factors / metabolism*

Substances

  • Alginates
  • Bacterial Proteins
  • Hexuronic Acids
  • Repressor Proteins
  • Virulence Factors
  • Flagellin
  • Glucuronic Acid