Intraesophageal administration of GS-nitroxide (JP4-039) protects against ionizing irradiation-induced esophagitis

In Vivo. 2010 Nov-Dec;24(6):811-9.

Abstract

Background/aim: this study evaluated esophageal radioprotection by the Gramicidin S (GS) derived-nitroxide, JP4-039, a mitochondrial targeting peptide-isostere covalently-linked to 4-amino-Tempo, delivered in a novel swallowed oil-based (F15) formulation.

Materials and methods: C57BL/6HNsd female mice received intraesophageal F15 formulation containing JP4-039 (4 mg/ml in 100 microl volumes) 10 minutes before 28 or 29 Gy upper body irradiation compared to MnSOD-PL (100 microl containing 100 microg plasmid) 24 hours prior to irradiation. Subgroups received 1 × 10(7) C57BL/6HNsd, GFP(+) male bone marrow cells intravenously 5 days after irradiation.

Results: JP4-039/F15 or MnSOD-PL increased survival compared to irradiated controls (p<0.0001 for either). Marrow injection further increased survival (p=0.0462 and 0.0351, respectively). Esophagi removed at 1, 3, 7, 14, 24, or 60 days showed bone marrow-derived cells in the esophagi.

Conclusion: intraesophageal GS-nitroxide radioprotection is mediated primarily through recovery of endogenous esophageal progenitor cells.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • Bone Marrow Transplantation
  • Cytoprotection
  • Drug Delivery Systems
  • Esophagitis / pathology
  • Esophagitis / prevention & control*
  • Esophagitis / therapy
  • Esophagus / drug effects*
  • Esophagus / pathology
  • Esophagus / radiation effects*
  • Female
  • Liposomes / therapeutic use
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Nitrogen Oxides / administration & dosage
  • Nitrogen Oxides / pharmacokinetics
  • Nitrogen Oxides / pharmacology*
  • Radiation Injuries, Experimental / pathology
  • Radiation Injuries, Experimental / prevention & control*
  • Radiation Injuries, Experimental / therapy

Substances

  • JP4-039
  • Liposomes
  • Nitrogen Oxides