Effect of pravastatin, an HMG CoA reductase inhibitor, and cholestyramine, a bile acid sequestrant, on lipoprotein particles defined by their apolipoprotein composition

Metabolism. 1990 Mar;39(3):269-73. doi: 10.1016/0026-0495(90)90046-f.

Abstract

This study compares the effects of cholestyramine (16 g/d) and pravastatin (40 mg/d) on lipoprotein particles defined by their apolipoprotein composition (Lp A-I, Lp A-II:A-I, Lp E:B, and Lp C-III:B). Analysis was performed after 4, 8, and 12 weeks of therapy. Low-density lipoprotein (LDL) cholesterol decreased by 25.1% to 35.0% with cholestyramine and 26.2% to 30.7% with pravastatin, while triglycerides decreased slightly with pravastatin therapy and increased slightly during cholestyramine administration. The fall in cholesterol was mainly due to a decrease in very-low-density lipoprotein (VLDL) and LDL cholesterol; high-density lipoprotein (HDL) cholesterol increased. Apolipoprotein B was reduced dramatically (by 21.7% to 30.5% with cholestyramine and 27.7% to 37.4% with pravastatin). No significant effect on apolipoproteins C-III and E was observed with cholestyramine, while pravastatin reduced these parameters slightly. Apolipoprotein A-I increased during therapy with both drugs, while apolipoprotein A-II was slightly decreased. Although the drugs had nearly the same effects on plasma lipids, their influence on lipoprotein particles defined by their apolipoprotein composition was substantially different. Lp A-II:A-I was increased by both drugs (+8.1% to +41.2% for cholestyramine and +7.2% to +32.6% for pravastatin). Lp A-I was also increased with both drugs, but cholestyramine had a more constant and pronounced effect than pravastatin (+15.1% to +21.7% for cholestyramine and +1.7% to +13.0% for pravastatin). Lp E:B and Lp C-III:B were consistently decreased by pravastatin (-10.2% to -36.5% for LP E:B and -7.2% to -20.9% for Lp C-III:B), while cholestyramine had variable effects on these particles.(ABSTRACT TRUNCATED AT 250 WORDS)

Publication types

  • Comparative Study

MeSH terms

  • Adult
  • Aged
  • Anticholesteremic Agents / therapeutic use*
  • Apolipoprotein A-I
  • Apolipoprotein A-II
  • Apolipoprotein C-III
  • Apolipoproteins / metabolism*
  • Apolipoproteins A / metabolism
  • Apolipoproteins B / metabolism
  • Apolipoproteins C / metabolism
  • Apolipoproteins E / metabolism
  • Bile Acids and Salts / metabolism
  • Cholesterol / metabolism
  • Cholesterol, HDL / metabolism
  • Cholesterol, LDL / metabolism
  • Cholesterol, VLDL / metabolism
  • Cholestyramine Resin / pharmacology
  • Cholestyramine Resin / therapeutic use*
  • Drug Evaluation
  • Female
  • Heptanoic Acids / pharmacology
  • Heptanoic Acids / therapeutic use*
  • Humans
  • Hypercholesterolemia / blood
  • Hypercholesterolemia / drug therapy*
  • Hypercholesterolemia / metabolism
  • Hyperlipoproteinemia Type II / blood
  • Hyperlipoproteinemia Type II / drug therapy*
  • Hyperlipoproteinemia Type II / metabolism
  • Lipid Metabolism
  • Lipoproteins / metabolism*
  • Male
  • Middle Aged
  • Naphthalenes / pharmacology
  • Naphthalenes / therapeutic use*
  • Pravastatin
  • Random Allocation
  • Triglycerides / metabolism

Substances

  • Anticholesteremic Agents
  • Apolipoprotein A-I
  • Apolipoprotein A-II
  • Apolipoprotein C-III
  • Apolipoproteins
  • Apolipoproteins A
  • Apolipoproteins B
  • Apolipoproteins C
  • Apolipoproteins E
  • Bile Acids and Salts
  • Cholesterol, HDL
  • Cholesterol, LDL
  • Cholesterol, VLDL
  • Heptanoic Acids
  • Lipoproteins
  • Naphthalenes
  • Triglycerides
  • Cholestyramine Resin
  • Cholesterol
  • Pravastatin