Temporoparietal hypometabolism in frontotemporal lobar degeneration and associated imaging diagnostic errors

Arch Neurol. 2011 Mar;68(3):329-37. doi: 10.1001/archneurol.2010.295. Epub 2010 Nov 8.

Abstract

Objective: To evaluate the cause of diagnostic errors in the visual interpretation of positron emission tomographic scans with fludeoxyglucose F 18 (FDG-PET) in patients with frontotemporal lobar degeneration (FTLD) and patients with Alzheimer disease (AD).

Design: Twelve trained raters unaware of clinical and autopsy information independently reviewed FDG-PET scans and provided their diagnostic impression and confidence of either FTLD or AD. Six of these raters also recorded whether metabolism appeared normal or abnormal in 5 predefined brain regions in each hemisphere-frontal cortex, anterior cingulate cortex, anterior temporal cortex, temporoparietal cortex, and posterior cingulate cortex. Results were compared with neuropathological diagnoses.

Setting: Academic medical centers.

Patients: Forty-five patients with pathologically confirmed FTLD (n=14) or AD (n=31).

Results: Raters had a high degree of diagnostic accuracy in the interpretation of FDG-PET scans; however, raters consistently found some scans more difficult to interpret than others. Unanimity of diagnosis among the raters was more frequent in patients with AD (27 of 31 patients [87%]) than in patients with FTLD (7 of 14 patients [50%]) (P=.02). Disagreements in interpretation of scans in patients with FTLD largely occurred when there was temporoparietal hypometabolism, which was present in 7 of the 14 FTLD scans and 6 of the 7 scans lacking unanimity. Hypometabolism of anterior cingulate and anterior temporal regions had higher specificities and positive likelihood ratios for FTLD than temporoparietal hypometabolism had for AD.

Conclusions: Temporoparietal hypometabolism in FTLD is common and may cause inaccurate interpretation of FDG-PET scans. An interpretation paradigm that focuses on the absence of hypometabolism in regions typically affected in AD before considering FTLD is likely to misclassify a significant portion of FTLD scans. Anterior cingulate and/or anterior temporal hypometabolism indicates a high likelihood of FTLD, even when temporoparietal hypometabolism is present. Ultimately, the accurate interpretation of FDG-PET scans in patients with dementia cannot rest on the presence or absence of a single region of hypometabolism but rather must take into account the relative hypometabolism of all brain regions.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Age of Onset
  • Aged
  • Alzheimer Disease / diagnostic imaging
  • Alzheimer Disease / metabolism
  • Alzheimer Disease / pathology
  • Brain / pathology
  • Diagnosis, Differential
  • Diagnostic Errors
  • Female
  • Fluorodeoxyglucose F18
  • Frontotemporal Lobar Degeneration / diagnostic imaging*
  • Frontotemporal Lobar Degeneration / metabolism*
  • Frontotemporal Lobar Degeneration / pathology
  • Humans
  • Image Processing, Computer-Assisted
  • Male
  • Middle Aged
  • Neuropsychological Tests
  • Observer Variation
  • Parietal Lobe / diagnostic imaging*
  • Parietal Lobe / metabolism*
  • Parietal Lobe / pathology
  • Positron-Emission Tomography
  • Radiopharmaceuticals
  • Reproducibility of Results
  • Temporal Lobe / diagnostic imaging*
  • Temporal Lobe / metabolism*
  • Temporal Lobe / pathology

Substances

  • Radiopharmaceuticals
  • Fluorodeoxyglucose F18