Synthetic RGDS-containing peptides of von Willebrand factor inhibit platelet adhesion to collagen

Thromb Haemost. 1990 Dec 28;64(4):589-93.

Abstract

We compared the effect of a synthetic dodecapeptide of residues 400-411 of the gamma chain of fibrinogen (gamma Fg 400-411) and of three synthetic peptides (15 to 18 aminoacids), of human von Willebrand Factor (vWF), containing the 1744-1747 Arg-Gly-Asp-Ser (RGDS) sequence, upon platelet adhesion to collagen in flowing blood. Both types of peptides are known to inhibit the binding of adhesive proteins to platelet membrane glycoprotein IIb/IIIa (GPIIb/IIIa). Collagen was coated onto plastic cover slips and exposed in parallel-plate perfusion chambers to reconstituted human blood at various shear rates for 5 min at 37 degrees C. At a shear rate of 2,600 s-1, RGDS peptides inhibited platelet adhesion to collagen in a dose-dependent manner and appeared to be more potent inhibitors than the gamma Fg 400-411 on a molar basis. No synergetic effect between RGDS and gamma Fg 400-411 peptides was observed. These results suggest that the RGDS peptides affect adhesion by inhibiting the GPIIb/IIIa-vWF interaction and confirm the involvement of this platelet receptor in vWF-mediated platelet adhesion to collagen at high shear rate.

MeSH terms

  • Amino Acid Sequence
  • Collagen
  • Hemostasis / physiology
  • Humans
  • Molecular Sequence Data
  • Oligopeptides / physiology*
  • Perfusion
  • Platelet Adhesiveness / physiology*
  • Platelet Count
  • Platelet Membrane Glycoproteins / physiology*
  • Stress, Mechanical
  • von Willebrand Factor / chemistry*

Substances

  • Oligopeptides
  • Platelet Membrane Glycoproteins
  • von Willebrand Factor
  • Collagen
  • arginyl-glycyl-aspartyl-serine