Role of interleukin-4 in the negative regulation of proliferation of chronic myelomonocytic leukemia cells

Ann N Y Acad Sci. 1991:628:156-64. doi: 10.1111/j.1749-6632.1991.tb17235.x.

Abstract

We studied the effects of interleukin-4 (IL-4) on the spontaneous proliferation of chronic myelomonocytic leukemia (CMMoL) cells in vitro to clarify whether IL-4 can act as a negative regulator of hematopoiesis. The results clearly show that IL-4 suppressed spontaneous DNA synthesis and colony formation by CMMoL cells in vitro. Colony formation by CMMoL cells was substantially suppressed by adding either an anti-IL-6 or anti-GM-CSF antibody to the colony assay system. This suppression was abrogated by the treatment of IL-4 with an anti-IL-4 antibody. Furthermore, the production of IL-6 or GM-CSF was markedly inhibited by adding IL-4 to the culture system. These observations indicate that IL-4 can act as a negative regulator for the autocrine growth of CMMoL cells by inhibiting their production of IL-6 and/or GM-CSF.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Aged, 80 and over
  • Cell Division / drug effects
  • DNA Replication / drug effects
  • Female
  • Granulocyte-Macrophage Colony-Stimulating Factor / biosynthesis
  • Hematopoiesis / physiology
  • Humans
  • In Vitro Techniques
  • Interleukin-4 / pharmacology*
  • Interleukin-6 / biosynthesis
  • Kinetics
  • Leukemia, Myelomonocytic, Chronic / blood
  • Leukemia, Myelomonocytic, Chronic / pathology*
  • Male
  • Middle Aged
  • Monocytes / drug effects
  • Monocytes / pathology*

Substances

  • Interleukin-6
  • Interleukin-4
  • Granulocyte-Macrophage Colony-Stimulating Factor