Her-2/neu expression in prostate adenocarcinoma: a systematic review and meta-analysis

J Urol. 2010 Sep;184(3):842-50. doi: 10.1016/j.juro.2010.04.077.

Abstract

Purpose: HER-2/neu has been linked to the clinical progression of hormone independent prostate cancer. We performed a meta-analysis to investigate the prognostic impact of HER-2/neu over expression in patients with prostate adenocarcinoma, and its correlation with other pathological and clinical variables.

Materials and methods: We searched the MEDLINE, Embase, CancerLit and ASCO abstract databases for published studies of HER-2/neu protein expression in primary prostate cancer tissue with a median followup of greater than 2 years and data on survival in patients with and without HER-2/neu over expression. We separately analyzed studies reporting HER-2/neu soluble receptor levels in patients with prostate cancer.

Results: We included 38 articles with a total of 5,976 patients. The overall RR of death in those with HER-2/neu over expression in the primary tumor was 1.63 (95% CI 1.47-1.82, p <0.0001). In the presence of over expression the recurrence RR was 1.87 (95% CI 1.59-2.21, p <0.0001). High HER-2/neu extracellular domain levels also correlated with death (RR 2.01, 95% CI 1.21-3.35, p = 0.007) and recurrence (RR 1.74, 95% CI 1.41-2.15, p <0.0001).

Conclusions: There is a consistent association of HER-2/neu over expression and Gleason less than 7 with a higher RR of death and recurrence in patients with prostate cancer. Further clinical trials should test the hypothesis that HER-2/neu is a marker of a clinically worse outcome in patients with prostate cancer and a potential target for therapy.

Publication types

  • Meta-Analysis
  • Review
  • Systematic Review

MeSH terms

  • Adenocarcinoma / blood
  • Adenocarcinoma / metabolism*
  • Biomarkers, Tumor / blood
  • Humans
  • Male
  • Prostatic Neoplasms / blood
  • Prostatic Neoplasms / metabolism*
  • Receptor, ErbB-2 / biosynthesis*
  • Receptor, ErbB-2 / blood

Substances

  • Biomarkers, Tumor
  • Receptor, ErbB-2