Identification of locus-specific DNA-binding factors for the regulation of HLA class-I genes in human colorectal cancer

Int J Cancer Suppl. 1991:6:131-7. doi: 10.1002/ijc.2910470724.

Abstract

The expression of HLA class-I mRNA in human colorectal cancer cell lines was studied. Locus-specific down-regulation of HLA class-I mRNA could be demonstrated in some of the human colorectal lines. This transcriptional suppression of HLA mRNA, however, was not a result of genetic alterations in the HLA structural genes. The transcription of the HLA class-I genes in the HLA-deficient cell lines could be induced by the addition of human recombinant gamma-IFN. In this report, we have employed these human colorectal cell lines to study locus-specific transcriptional regulation of HLA class-I gene expression. Our results demonstrate that the locus-specific suppression of HLA gene expression in human colorectal cell lines is mediated by the loss of certain DNA-binding transcription factors which act in a locus-specific manner. Our conclusion is further supported by experiments which showed that exogenously introduced HLA-CAT DNA constructs were also regulated in a locus-specific fashion when assayed by in vitro functional assays using these human colorectal cell lines.

MeSH terms

  • Adenocarcinoma / genetics
  • Adenocarcinoma / immunology*
  • Base Sequence
  • Binding Sites
  • Cell Line
  • Cell Nucleus / metabolism
  • Chloramphenicol O-Acetyltransferase / genetics
  • Chloramphenicol O-Acetyltransferase / metabolism
  • Colonic Neoplasms / genetics
  • Colonic Neoplasms / immunology*
  • DNA-Binding Proteins / isolation & purification
  • DNA-Binding Proteins / metabolism*
  • Gene Expression Regulation, Neoplastic*
  • Genes, MHC Class I*
  • HLA-A Antigens / genetics
  • HLA-B Antigens / genetics
  • Humans
  • Molecular Sequence Data
  • Oligonucleotide Probes
  • RNA, Messenger / analysis
  • RNA, Messenger / genetics
  • Rectal Neoplasms / genetics
  • Rectal Neoplasms / immunology*
  • Transcription, Genetic
  • Transfection

Substances

  • DNA-Binding Proteins
  • HLA-A Antigens
  • HLA-B Antigens
  • Oligonucleotide Probes
  • RNA, Messenger
  • Chloramphenicol O-Acetyltransferase