Effect of neuropeptide Y on jejunal water and ion transport in humans

Gastroenterology. 1991 Aug;101(2):325-30. doi: 10.1016/0016-5085(91)90007-8.

Abstract

Neuropeptide Y is a neurotransmitter in enteric and postganglionic sympathetic neurons. In animal models of intestinal water and ion transport, neuropeptide Y decreases stimulated secretion but has no consistent effect on basal transport. In the present study, the effect of neuropeptide Y on jejunal water and electrolyte transport in healthy volunteers was investigated under basal conditions and during intestinal secretion induced by intraluminal administration of prostaglandin E2. The triple-lumen tube technique was used for perfusion of the small intestine with a plasmalike electrolyte solution containing polyethylene glycol as a nonabsorbable volume marker. After an initial control period (saline IV) neuropeptide Y was administered IV at a dose of 400 pmol.kg-1.h-1. Neuropeptide Y significantly increased net absorption of water, sodium, potassium, and chloride under basal conditions. The peptide significantly reduced the secretion of these electrolytes induced by an intraluminal prostaglandin E2 concentration of 5 mumol/L and reduced net water secretion by 36%. The results of the current study suggest that neuropeptide Y can change intestinal water and ion transport from secretion toward absorption.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Bicarbonates / metabolism
  • Chlorides / metabolism
  • Dinoprostone / pharmacology
  • Female
  • Humans
  • Intestinal Absorption / drug effects*
  • Jejunum / drug effects
  • Jejunum / metabolism*
  • Male
  • Neuropeptide Y / blood
  • Neuropeptide Y / pharmacology*
  • Potassium / metabolism
  • Sodium / metabolism
  • Water / metabolism
  • Water-Electrolyte Balance / drug effects*

Substances

  • Bicarbonates
  • Chlorides
  • Neuropeptide Y
  • Water
  • Sodium
  • Dinoprostone
  • Potassium