Dendritic cells loaded with tumor B cells elicit broad immunity against murine gammaherpesvirus 68 but fail to prevent long-term latency

J Virol. 2010 Sep;84(17):8975-9. doi: 10.1128/JVI.00571-10. Epub 2010 Jun 30.

Abstract

It is still unknown whether a noninfectious gammaherpesvirus vaccine is able to prevent or reduce virus persistence. This led us to use dendritic cells loaded with tumor B cells as a vaccine approach for the murine gammaherpesvirus 68 (gammaHV68) model of infection. Dendritic cells loaded with UV-irradiated latently infected tumor B cells induce broad, strong, and long-lasting immunity against gammaHV68. Dendritic cell vaccination prevents the enlargement of lymph nodes and severely limits acute infection and early latency but does not prevent gammaHV68 from establishing long-term latency. Our findings support the concept that attenuated viruses may be the best vaccine option for preventing gammaherpesvirus persistence.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • B-Lymphocytes / immunology*
  • B-Lymphocytes / virology
  • CD4-Positive T-Lymphocytes / immunology
  • CD8-Positive T-Lymphocytes / immunology
  • Cancer Vaccines / administration & dosage
  • Cancer Vaccines / immunology*
  • Cells, Cultured
  • Dendritic Cells / immunology*
  • Dendritic Cells / virology
  • Gammaherpesvirinae / genetics
  • Gammaherpesvirinae / immunology
  • Gammaherpesvirinae / physiology*
  • Herpesviridae Infections / immunology*
  • Herpesviridae Infections / virology
  • Humans
  • Lymphoma, B-Cell / immunology
  • Mice
  • Mice, Inbred BALB C
  • Vaccination
  • Virus Latency*

Substances

  • Cancer Vaccines