[The expression of TWEAKR/Fn14 in Rheumatoid arthritis fibroblast-like synoviocytes]

Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi. 2010 Jun;26(6):575-7.
[Article in Chinese]

Abstract

Aim: To investigate the mechanism of TWEAK on Rheumatoid arthritis fibroblast-like synoviocytes (RA FLS) activation and articular destruction by analyzing expression of TWEAKR/Fn14 on RA FLS.

Methods: FLS was co-cultured with rhTWEAK, with TNF-alpha or IL-1beta as a synergistic stimulator. Expression of TWEAKR/Fn14 protein on RA FLS was detected by Immunocytochemistry and Western blotting. The fn14 mRNA was measured by RT-PCR.

Results: TWEAKR/Fn14 was expressed on the cell membrane and in the cytoplasm of RA FLS. TWEAKR/Fn14 mRNA and protein induced by 100 microg/L TWEAK were higher than the control group. TNF-alpha and IL-1beta had synergistic effect on expression of TWEAKR/Fn14 in RA FLS.

Conclusion: TWEAKR/Fn14 was expressed on the cell membrane and in the cytoplasm of RA FLS, and recombinant TWEAK further enhanced the TWEAKR/Fn14 expression. TNF-alpha and IL-1beta had synergistic effect on the biological activity of TWEAK by increasing the expression of TWEAKR/Fn14 on RA FLS.

MeSH terms

  • Animals
  • Arthritis, Rheumatoid / metabolism*
  • Fibroblasts / cytology
  • Fibroblasts / drug effects
  • Fibroblasts / metabolism*
  • Humans
  • Immunohistochemistry
  • Interleukin-1beta / pharmacology
  • Mice
  • Receptors, Tumor Necrosis Factor / genetics
  • Receptors, Tumor Necrosis Factor / metabolism*
  • Reverse Transcriptase Polymerase Chain Reaction
  • Synovial Membrane / cytology
  • Synovial Membrane / metabolism*
  • TWEAK Receptor
  • Tumor Necrosis Factor-alpha / pharmacology

Substances

  • Interleukin-1beta
  • Receptors, Tumor Necrosis Factor
  • TNFRSF12A protein, human
  • TWEAK Receptor
  • Tnfrsf12a protein, mouse
  • Tumor Necrosis Factor-alpha