Influence of dietary-induced copper and manganese deficiency on ozone-induced changes in lung and liver antioxidant systems

Toxicol Lett. 1991 Jun;57(1):81-90. doi: 10.1016/0378-4274(91)90122-m.

Abstract

The influence of an animal's copper (Cu) and manganese (Mn) status on its response to ozone was investigated in weanling mice. Control, Cu-deficient and Mn-deficient mice were exposed continuously to 1.2 ppm O3 or filtered air for 7 days. In control mice, ozone exposure resulted in higher lung activities of CuZnSOD, MnSOD and GPx. In contrast, Mn-deficient mice did not display increases in lung MnSOD, CuZnSOD or GPx activities following ozone exposure. Similarly, ozone-induced increases in lung CuZn-SOD and MnSOD activities were not observed in Cu-deficient mice, although lung GPx activity was increased in these mice relative to their air-breathing controls. These results show that an animal's Cu and Mn status can influence its response to ozone, and the data suggest that Cu- and Mn-deprived animals may be more susceptible to long-term or repetitive ozone exposure.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Body Weight / drug effects
  • Copper / deficiency*
  • Diet*
  • Female
  • Glutathione / metabolism
  • Glutathione Peroxidase / metabolism
  • Liver / drug effects*
  • Liver / metabolism
  • Lung / drug effects*
  • Lung / metabolism
  • Manganese / deficiency*
  • Mice
  • Organ Size / drug effects
  • Ozone / toxicity*
  • Superoxide Dismutase / metabolism

Substances

  • Manganese
  • Ozone
  • Copper
  • Glutathione Peroxidase
  • Superoxide Dismutase
  • Glutathione