Abstract
Introduction:
The antimicrobial peptide LL37 has multiple functions, such as the induction of angiogenesis and migration. Pulp cell migration is a key phenomenon in the early stage of pulp-dentin complex regeneration. In this study, we examined the effect of LL37 on the migration of human pulp (HP) cells.
Methods:
HP cells at the sixth passage were exposed to LL37. The migration of HP cells was assessed by a wound-healing assay. The phosphorylation of epidermal growth factor receptor (EGFR) and c-Jun N-terminal kinase (JNK) was analyzed by immunoblotting.
Results:
LL37 as well as heparin binding (HB)-EGF, which is an agonist of EGFR, induced HP cell migration. LL37 increased the level of phosphorylated EGFR. An anti-EGFR antibody, an EGFR tyrosine kinase inhibitor, and a JNK inhibitor abolished the migration induced by both LL37 and HB-EGF. Furthermore, the two peptides increased the levels of phosphorylated JNK.
Conclusions:
LL37 activates EGFR and JNK to induce HP cell migration, and it may contribute to enhancing the regeneration of pulp-dentin complexes.
Copyright 2010 American Association of Endodontists. Published by Elsevier Inc. All rights reserved.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Adenosine Triphosphate / analogs & derivatives
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Adenosine Triphosphate / pharmacology
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Anthracenes / pharmacology
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Antimicrobial Cationic Peptides
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Calcium-Calmodulin-Dependent Protein Kinases / antagonists & inhibitors
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Cathelicidins / pharmacology*
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Cell Movement / drug effects
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Cells, Cultured
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Dental Pulp / cytology
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Dental Pulp / drug effects*
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Endodontics
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Enzyme Inhibitors / pharmacology
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ErbB Receptors / agonists
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ErbB Receptors / antagonists & inhibitors
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ErbB Receptors / drug effects
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Flavonoids / pharmacology
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Heparin / pharmacology
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Heparin-binding EGF-like Growth Factor
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Humans
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Intercellular Signaling Peptides and Proteins / pharmacology
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JNK Mitogen-Activated Protein Kinases / antagonists & inhibitors
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JNK Mitogen-Activated Protein Kinases / drug effects
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Phosphorylation
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Protein Tyrosine Phosphatases / antagonists & inhibitors
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Purinergic P2 Receptor Agonists
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Quinazolines
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Receptors, Cell Surface / drug effects
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Receptors, Purinergic P2X
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Regenerative Medicine
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Tissue Engineering / methods
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Tyrphostins / pharmacology
Substances
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Anthracenes
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Antimicrobial Cationic Peptides
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Cathelicidins
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Enzyme Inhibitors
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Flavonoids
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HBEGF protein, human
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Heparin-binding EGF-like Growth Factor
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Intercellular Signaling Peptides and Proteins
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Purinergic P2 Receptor Agonists
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Quinazolines
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Receptors, Cell Surface
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Receptors, Purinergic P2X
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Tyrphostins
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RTKI cpd
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pyrazolanthrone
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3'-O-(4-benzoyl)benzoyladenosine 5'-triphosphate
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Adenosine Triphosphate
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Heparin
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ErbB Receptors
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Calcium-Calmodulin-Dependent Protein Kinases
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JNK Mitogen-Activated Protein Kinases
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Protein Tyrosine Phosphatases
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2-(2-amino-3-methoxyphenyl)-4H-1-benzopyran-4-one