Refining the phenotype associated with MEF2C haploinsufficiency

Clin Genet. 2010 Nov;78(5):471-7. doi: 10.1111/j.1399-0004.2010.01413.x.

Abstract

Recently, submicroscopic deletions of the 5q14.3 region have been described in patients with severe mental retardation (MR), stereotypic movements, epilepsy and cerebral malformations. Further delineation of a critical region of overlap in these patients pointed to MEF2C as the responsible gene. This finding was further reinforced by the identification of a nonsense mutation in a patient with a similar phenotype. In brain, MEF2C is essential for early neurogenesis, neuronal migration and differentiation. Here we present two additional patients with severe MR, autism spectrum disorder and epilepsy, carrying a very small deletion encompassing the MEF2C gene. This finding strengthens the role of this gene in severe MR, and enables further delineation of the clinical phenotype.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Child Development Disorders, Pervasive / genetics
  • Child, Preschool
  • Epilepsies, Myoclonic / genetics
  • Haploinsufficiency
  • Humans
  • Infant
  • Intellectual Disability / genetics
  • MADS Domain Proteins / genetics*
  • MEF2 Transcription Factors
  • Male
  • Myogenic Regulatory Factors / genetics*
  • Phenotype
  • Sequence Deletion

Substances

  • MADS Domain Proteins
  • MEF2 Transcription Factors
  • MEF2C protein, human
  • Myogenic Regulatory Factors