Killing effect of interleukin-13 receptor alpha 2 (IL-13Ralpha2) sensitized DC-CTL cells on human glioblastoma U251 cells

Cell Immunol. 2010;263(2):172-5. doi: 10.1016/j.cellimm.2010.03.013. Epub 2010 Mar 27.

Abstract

Recent studies show that IL-13Ralpha2, a brain tumor-associated antigen for IL-13, may play a role in immunotherapy for glioblastoma. Thus, we stimulated the lymphocyte by monocyte-derived dendritic cells (DCs). The DCs were pulsed with IL-13Ralpha2 in vitro and then co-cultured with lymphocytes. After inducing cytotoxic T cells (CTLs) and co-culturing with U251 cells for 24 h in 96 wells, Cell Count Kit-8 (CCK-8) was added to every well equally. The optical density (OD) value was detected and recorded after 2 h. The DCs efficiently presented the antigen to the CTLs, resulting in CTLs activation and proliferation. The induced CTLs showed specific cytotoxic against U251 cells (P<0.01). The results demonstrated that IL-13Ralpha2 induced CTLs could kill glioma U251 in vitro, which suggests that IL-13 Ralpha2 might have such an impact in vivo and thus recombinant IL-13Ra2 protein might be used as an anti-tumor vaccine, providing a promising new strategy for the treatment of brain malignant gliomas.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Brain Neoplasms*
  • Cell Line, Tumor
  • Cell Proliferation
  • Cells, Cultured
  • Dendritic Cells / immunology*
  • Glioblastoma*
  • Humans
  • Interleukin-13 Receptor alpha2 Subunit / immunology*
  • Lymphocyte Activation / immunology
  • T-Lymphocytes, Cytotoxic / cytology*
  • T-Lymphocytes, Cytotoxic / immunology*

Substances

  • Interleukin-13 Receptor alpha2 Subunit