Nitric oxide-induced IL-8 expression is mediated by NF-kappaB and AP-1 in gastric epithelial AGS cells

J Physiol Pharmacol. 2009 Dec:60 Suppl 7:101-6.

Abstract

Inducible nitric oxide synthase (iNOS) and interleukin-8 (IL-8) mediate gastric inflammation. Nitric oxide (NO) produced by iNOS may activate oxidant-sensitive transcription factors. There are the binding sites for NF-kappaB, AP-1, and C/EBP (CCAAT/enhancer binding protein) in the promoter regions of IL-8 gene. The present study aims to investigate whether NO donors, SIN-1 and NOC-18, activate oxidant-sensitive transcription factors NF-kappaB and AP-1 as well as C/EBP to induce IL-8 expression in gastric epithelial AGS cells. Gastric epithelial AGS cells were treated with NO donors, SIN-1 and NOC-18. mRNA expression and protein level of IL-8 in the medium were determined. Nitrite level in the medium and DNA binding activities of NF-kappaB, AP-1, and C/EBP were assessed. NO donors induced the increase in the levels of IL-8 and nitrite in the medium as well as mRNA expression of IL-8 in AGS cells time-dependently. The induction of IL-8 by NO donors was accompanied with the activation of NF-kappaB and AP-1 but not C/EBP in AGS cells.

Conclusion: Large amount of NO, which may be produced by iNOS, may induce the activation of NF-kappaB and AP-1 and the expression of IL-8 in gastric epithelial cells.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Anti-Ulcer Agents / pharmacology
  • CCAAT-Enhancer-Binding Proteins / metabolism
  • Cell Line
  • DNA-Binding Proteins / metabolism
  • Electrophoretic Mobility Shift Assay
  • Epithelial Cells
  • Gastric Mucosa / drug effects*
  • Gene Expression Regulation / drug effects
  • Genes, Reporter
  • Humans
  • Interleukin-8 / genetics
  • Interleukin-8 / metabolism*
  • Molsidomine / analogs & derivatives
  • Molsidomine / pharmacology
  • NF-kappa B / metabolism*
  • Nitric Oxide / analysis
  • Nitric Oxide / physiology*
  • Nitric Oxide Donors / pharmacology
  • Nitroso Compounds / pharmacology
  • RNA, Messenger / metabolism
  • Time Factors
  • Transcription Factor AP-1 / metabolism*

Substances

  • Anti-Ulcer Agents
  • CCAAT-Enhancer-Binding Proteins
  • DNA-Binding Proteins
  • Interleukin-8
  • NF-kappa B
  • NOC 18
  • Nitric Oxide Donors
  • Nitroso Compounds
  • RNA, Messenger
  • Transcription Factor AP-1
  • Nitric Oxide
  • linsidomine
  • Molsidomine