Noradrenaline release from the rat heart during anoxia: effects of changes in extracellular sodium concentration and inhibition of sodium uptake mechanisms

Clin Exp Pharmacol Physiol. 1991 Jan;18(1):43-6. doi: 10.1111/j.1440-1681.1991.tb01375.x.

Abstract

1. Anoxic perfusion of the isolated rat heart releases noradrenaline in the absence of sympathetic nerve fibre stimulation. 2. Anoxic noradrenaline release is enhanced by reducing the extracellular Na+ concentration, consistent with the proposal that such release occurs by carrier-mediated efflux. 3. Release is also enhanced by lignocaine but inhibited by amiloride and ethylisopropylamiloride, suggesting that sodium entry into adrenergic nerve terminals during anoxia occurs by Na+/H+ (and possibly Na+/Ca2+) exchange.

MeSH terms

  • Amiloride / analogs & derivatives
  • Amiloride / pharmacology
  • Animals
  • Extracellular Space / metabolism*
  • Hypoxia / metabolism*
  • In Vitro Techniques
  • Lidocaine / pharmacology
  • Male
  • Myocardium / metabolism*
  • Norepinephrine / metabolism*
  • Rats
  • Rats, Inbred Strains
  • Sodium / metabolism*

Substances

  • Amiloride
  • Lidocaine
  • Sodium
  • ethylisopropylamiloride
  • Norepinephrine