Abstract
Platinum complexes with N,N'-bis(1-hydroxybut-2-yl)ethylenediamine, [PtCl2(ethambutol)] were prepared and the biological activity of three isomers [with (-), (+) and (+/-) ethambutol, respectively] investigated. All species interact with the Bam HI and Ava I recognition sequences showing a binding preference for GC rich sequences of DNA. The complex which showed the greatest interaction with adjacent guanines, [PtCl2[+/-)ethambutol)] was also found to be the most mutagenic of the three. On the other hand, only [PtCl2[+)ethambutol)] had a considerable antitumour activity against both P388 leukaemia and Lewis lung carcinoma, and this was not correlated either with restriction enzyme blocking activity or with mutagenicity.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Animals
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Antineoplastic Agents / therapeutic use*
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Binding Sites
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Cisplatin / pharmacology*
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Ethambutol / analogs & derivatives*
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Ethambutol / chemical synthesis
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Ethambutol / pharmacology*
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Ethambutol / therapeutic use
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Leukemia P388 / drug therapy*
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Lung Neoplasms / drug therapy*
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Male
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Mice
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Mice, Inbred C57BL
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Mice, Inbred DBA
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Mice, Inbred Strains
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Molecular Structure
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Mutagenicity Tests
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Mutagens / pharmacology*
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Organoplatinum Compounds / chemical synthesis
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Organoplatinum Compounds / pharmacology*
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Organoplatinum Compounds / therapeutic use
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Plasmids*
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Restriction Mapping
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Salmonella typhimurium / drug effects
Substances
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Antineoplastic Agents
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Mutagens
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Organoplatinum Compounds
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platinum-ethambutol
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Ethambutol
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Cisplatin