[Effect of cinobufagin on nuclear factor-kappaB pathway in HepG2 cells]

Nan Fang Yi Ke Da Xue Xue Bao. 2010 Jan;30(1):137-9.
[Article in Chinese]

Abstract

Objective: To investigate the effect of cinobufagin on nuclear factor-kappaB (NF-kappaB) pathway of liver cancer cell line HepG2.

Methods: Dual-luciferase cis-reporting system was used to detect the relative value of pNF-kappaB-TA-luc upon tumor necrosis factor-alpha (TNF-alpha) stimulation of NF-kappaB pathway. Western blotting was used to detect the protein level of NF-kappaB p65, and RT-PCR was used to detect the gene transcription level of intercellular adhesion molecule-1 (ICAM-1), a target downstream gene of NF-kappaB.

Results: At the concentration of 0.25 and 0.5 microg/ml, cinobufagin significantly lowered the relative value of luciferase (P<0.05). The results of Western blotting showed that cinobufagin significantly suppressed the protein expression of NF-kappaB p65. The transcription level of ICAM-1 was reduced by different doses of cinobufagin.

Conclusion: The anti-cancer effect of cinobufagin may be related to its activity in inhibiting the activation of NF-kappaB pathway.

Publication types

  • English Abstract

MeSH terms

  • Antineoplastic Agents / pharmacology*
  • Bufanolides / pharmacology*
  • Hep G2 Cells
  • Humans
  • Intercellular Adhesion Molecule-1 / genetics
  • Intercellular Adhesion Molecule-1 / metabolism
  • Materia Medica / pharmacology
  • NF-kappa B / drug effects*
  • Signal Transduction / drug effects*
  • Transcription Factor RelA / genetics
  • Transcription Factor RelA / metabolism

Substances

  • Antineoplastic Agents
  • Bufanolides
  • Materia Medica
  • NF-kappa B
  • Transcription Factor RelA
  • Intercellular Adhesion Molecule-1
  • cinobufagin