Cell and receptor requirements for streptococcal pyrogenic exotoxin T-cell mitogenicity

Infect Immun. 1991 Mar;59(3):1210-4. doi: 10.1128/iai.59.3.1210-1214.1991.

Abstract

Streptococcal pyrogenic exotoxins (SPEs) A, B, and C, like other members of the pyrogenic toxin family, are able to cause toxic shock-like syndromes. One of the major properties of these toxins is the ability to induce T-cell proliferation. Characterization of T cell mitogenicity associated with SPEs A, B, and C was undertaken. SPEs A, B, and C were mitogenic for C57BL10/SnJ and BALB/cWAT T cells, with activities differing in intensity depending on the mouse strain and toxin employed. SPE-induced, T-cell-proliferative activity was dependent on class II major histocompatibility complex molecules expressed on antigen-presenting cells. The abilities of SPEs A, B, and C to preferentially stimulate murine cells with certain T-cell receptor V beta s were investigated by fluorescence-activated cell sorter analysis. SPE A preferentially activated T cells expressing V beta 8 but not V beta 3, 6, or 11, while SPEs B and C preferentially stimulated T cells which did not express any of the tested V beta s.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Antibodies, Monoclonal
  • Antigen-Presenting Cells / drug effects
  • Antigen-Presenting Cells / immunology
  • Bacterial Proteins*
  • Exotoxins / metabolism
  • Exotoxins / toxicity*
  • Histocompatibility Antigens Class II / biosynthesis
  • Lymphocyte Activation / drug effects
  • Membrane Proteins*
  • Mice
  • Mice, Inbred BALB C
  • Mice, Inbred C57BL
  • Mitogens / metabolism
  • Mitogens / toxicity*
  • Receptors, Antigen, T-Cell / drug effects*
  • T-Lymphocytes / drug effects*
  • T-Lymphocytes / immunology

Substances

  • Antibodies, Monoclonal
  • Bacterial Proteins
  • Exotoxins
  • Histocompatibility Antigens Class II
  • Membrane Proteins
  • Mitogens
  • Receptors, Antigen, T-Cell
  • SpeA protein, Streptococcus pyogenes
  • erythrogenic toxin