Crossed cerebellar diaschisis in status epilepticus

Neurocrit Care. 2010 Feb;12(1):88-90. doi: 10.1007/s12028-009-9312-y.

Abstract

Background: Crossed cerebellar diaschisis (CCD) has been reported on positron-emission tomography and single-photon emission computed tomography of stroke patients. Rarely it has been described with brain diffusion-weighted MRI (DWI) of status epilepticus (SE).

Methods: Case report.

Results: A 53-year-old woman was found unresponsive after cocaine use. A diagnostic electroencephalogram was consistent with ictal SE. A brain DWI showed reduced diffusion in the left temporo-parietal and occipital cortexes, the left thalamus and the right cerebellum. The DWI changes did not correspond to a vascular territory and were attributed to seizure activity and secondary CCD. A 2-week follow-up DWI showed interval near-complete resolution of the diffusion changes. CCD in SE may represent injury caused by excessive neuronal transmission from prolonged excitatory synaptic activity via the cortico-pontine-cerebellar pathway. Alternatively, it may be a result of interruption of the cortico-pontine-cerebellar pathway with loss of cortical inhibitory input.

Conclusion: This case documents CCD during SE, providing further evidence of contralateral cerebellar involvement with a supratentorial epileptiform focus.

Publication types

  • Case Reports

MeSH terms

  • Brain Ischemia / chemically induced
  • Brain Ischemia / diagnosis*
  • Brain Ischemia / physiopathology
  • Cerebellar Diseases / chemically induced*
  • Cerebellar Diseases / diagnosis*
  • Cerebellar Diseases / physiopathology
  • Cerebellum / blood supply*
  • Cocaine-Related Disorders / diagnosis*
  • Cocaine-Related Disorders / physiopathology
  • Diffusion Magnetic Resonance Imaging*
  • Dominance, Cerebral / drug effects
  • Dominance, Cerebral / physiology*
  • Electroencephalography
  • Energy Metabolism / drug effects
  • Energy Metabolism / physiology*
  • Female
  • Humans
  • Middle Aged
  • Neurons / drug effects
  • Neurons / physiology
  • Status Epilepticus / chemically induced*
  • Status Epilepticus / diagnosis*
  • Status Epilepticus / physiopathology