Interleukin-6 -174 G/C promoter polymorphism and nasal polyposis

Rhinology. 2009 Dec;47(4):400-4. doi: 10.4193/Rhin08.226.

Abstract

Introduction: Nasal polyposis is a chronic disease with unknown etiopathogenesis, although inflammatory mechanisms seem to play a role. One of several inflammatory mediators linked to nasal polyposis is Interleukin-6, which has a single nucleotide polymorphism -174 G/C that seems to promote an inflammatory reaction.

Objective: To compare the prevalence of the -174 G/C single nucleotide polymorphism between a group of patients with nasal polyposis and a control group.

Method: Cross-sectional study with two groups (thirty two patients with nasal polyposis and fifty five controls) to investigate the -174 G/C polymorphism in blood samples. Asthma, aspirin intolerance and atopy were main exclusion criteria. IL-6 genotyping was performed using the PCR method with forward primer 5'-ATGCCAAGTGCTGAGTCACTA-3' and reverse primer 5'-GGAAAATCCCACATTTGATA-3', amplifying a 226-bp DNA fragment that contained the - 174 position. The amplified fragment can be cleaved by restriction enzyme NlaIII when the -174 position presented the C allele in two fragments of 117 and 109-bp, visualized by electrophoresis, classifying participants in GG, GC and CC.

Results: In the nasal polyposis group, 65.62% of the patients had the GG genotype, while in the control group, 41.82% had two G alleles, a statistically significant difference, with an odds ratio of 2.65.

Conclusion: The -174 GG genotype was found more frequently in nasal polyposis patients than in controls, when asthma, aspirin intolerance and atopy were excluded.

MeSH terms

  • Adult
  • Aged
  • Cross-Sectional Studies
  • DNA Fragmentation
  • Electrophoresis, Agar Gel
  • Female
  • Genotype
  • Humans
  • Interleukin-6 / genetics*
  • Male
  • Middle Aged
  • Nasal Polyps / genetics*
  • Polymorphism, Single Nucleotide / genetics*

Substances

  • Interleukin-6